Innate response activator B cells protect against microbial sepsis.

نویسندگان

  • Philipp J Rauch
  • Aleksey Chudnovskiy
  • Clinton S Robbins
  • Georg F Weber
  • Martin Etzrodt
  • Ingo Hilgendorf
  • Elizabeth Tiglao
  • Jose-Luiz Figueiredo
  • Yoshiko Iwamoto
  • Igor Theurl
  • Rostic Gorbatov
  • Michael T Waring
  • Adam T Chicoine
  • Majd Mouded
  • Mikael J Pittet
  • Matthias Nahrendorf
  • Ralph Weissleder
  • Filip K Swirski
چکیده

Recognition and clearance of a bacterial infection are a fundamental properties of innate immunity. Here, we describe an effector B cell population that protects against microbial sepsis. Innate response activator (IRA) B cells are phenotypically and functionally distinct, develop and diverge from B1a B cells, depend on pattern-recognition receptors, and produce granulocyte-macrophage colony-stimulating factor. Specific deletion of IRA B cell activity impairs bacterial clearance, elicits a cytokine storm, and precipitates septic shock. These observations enrich our understanding of innate immunity, position IRA B cells as gatekeepers of bacterial infection, and identify new treatment avenues for infectious diseases.

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عنوان ژورنال:
  • Science

دوره 335 6068  شماره 

صفحات  -

تاریخ انتشار 2012